Departments & Divisions
Currently seeking M.S. & Ph.D. students.
Director, Center for Mucosal & Microbiome Biology
Dr. Mansour M. Zadeh is a cellular and molecular immunologist. Dr. Zadeh is a Distinguished Professor in Medicine and the Director of The Center for Mucosal & Microbiome Biology at the UT Health San Antonio - Joe R. & Teresa Lozano Long School of Medicine. His laboratory’s work is tightly focused on the interaction of gut microbiome and the associated metabolites (e.g., vitamin B12) with the hosts during steady states and proinflmammatory diseases such as inflammatory bowel disease (IBD). Dr. Zadeh’s goal is to robustly establish an advanced scientific platform in cellular and molecular mucosal immunology and microbiome research by supporting the education of both basic and clinical scientists. Dr. Zadeh’s research and educational endeavors are geared toward creating an environment that facilitates a commitment to innovative research excellence and patient care.
2021 UT System Faculty STARs Award, San Antonio, TX
2020 Univ of Florida Research Foundation Professor
2017 Univ of Florida Professorship Award for Research Excellence
2017 Pfizer Award for Research Excellence
2017 Highest Distinction Award by Anderson Scholar Faculty Honoree, College of Liberal Arts and Sciences
2016 Technology Award by Office of Technology Licensing, Univ of Florida
2013 Superior Accomplishment Award in the Faculty Category, Univ of Florida, Gainesville, FL
Dr. Mansour Zadeh is an immunologist with over 27 years of expertise in the fields of inflammation, microbiome, systemic/mucosal immunology, infection, and vaccine delivery. Dr. Zadeh’s research is focused on elucidating beneficial gut microbes, their unique properties and role in the induction of stimulatory or regulatory signals in innate cells and T lymphocytes, contributing directly to further proinflammation or regulation of inflammatory diseases (IBD, NEC). His work is tightly entwined with generating novel therapies for intestinal disorders (e.g. NEC) using beneficial bacterium to significantly regulate induced pathogenic inflammation in sufferers.